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1.
Motriz rev. educ. fís. (Impr.) ; 22(2): 3-8, Apr.-June 2016. Ilus
Article in English | LILACS | ID: lil-781525

ABSTRACT

Alterations in the heart rate recovery and heart rate variability have been associated with greater risk of mortality and early prognosis of cardiac diseases. Thus, strategies for assessing autonomic nervous system and its modulation to the heart are crucial for preventing cardiovascular events in healthy subjects as well as in cardiac patients. In this review, an update of studies examining heart rate variability (HRV) and its use as indicator of cardiac autonomic modulation will be discussed. It will be described the indexes and methods of analysis and its applicability and the effects of exercise training on HRV and heart rate recovery in different population


Subject(s)
Humans , Autonomic Nervous System , Exercise , Heart Rate
2.
Motriz rev. educ. fís. (Impr.) ; 22(1): 12-17, Jan.-Mar. 2016. tab, graf
Article in English | LILACS | ID: lil-776618

ABSTRACT

The aim of this study was to evaluate the effect aerobic exercise training on fat pad mass, adipocyte size, leptin release and insulin sensitivity in rats fed with high fat-palatable diet. Twenty-four male Wistar rats (250-260g) were divided into four groups: sedentary control (CTR/SD), trained control (CTR/TR), obese sedentary (OB/SD) and obese trained (OB/TR). Obese groups were fed with high fat-palatable diet (27% of fat) and control groups fed with AIN-93. Our results showed that aerobic exercise training was effective to reduce body weight and epididymal fat mass in CTR/TR and OB/TR. Insulin and glucose levels were increased in OB/TR compared with OB/SD. Aerobic exercise training reduced the average area of adipocytes in CTR/TR and OB/TR and it was associated with reduced plasma insulin and leptin. In conclusion, 7-week aerobic exercise training reduces adipocyte area and improves insulin sensitivity and leptin levels in high fat-palatable diet-fed Wistar rats.


Subject(s)
Animals , Male , Rats , Dietary Fats , Exercise , Hypoglycemic Agents , Insulin , Leptin
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